French explorer, Jean-Baptiste Le Moyne de Bienville, and his brother, Pierre Le Moyne d'Iberville, first entered Bayou St. John from Lake Pontchartrain in 1699, and the rest, as they say, is history. The Mississippi River is closely identified with the story of the city's settlement, but there's no way to exaggerate the importance of Bayou St. John, along with its portage trail, to the development of the area. The Bayou connected Lake Pontchartrain (and its access to the Gulf of Mexico) with the Mississippi River. Native Americans had used the waterway, which they called "Bayouk Choupic," since pre-Columbian times. In fact, Bienville chose the location of "Nouvelle Orleans" because of the site's proximity to the Bayou, as well as, the sharp crescent in the River there, which he believed would help protect the settlement from storms. The construction of dwellings at the site on the Mississippi River began in 1718, but concessions had already been granted along Bayou St. John as early as 1708, and it wasn't long before all of the land along the length of the Bayou held dwellings ranging from small huts to substantial homes. Today, Bayou St. John is a recreational waterway, much appreciated for its beauty and its atmosphere, as it flows peacefully through the heart of the city.
In This Issue
Vitamins Offer Hope For Alzheimers
Receptor May Aid Spread Of Alzheimers & Parkinson's In Brain
Great Grams Inspires Teens To Hunt For Alzheimer's Cure
Remarkable Case Of A Canadian Sculptor
Alzheimer's Store Featured Product
Recipe Of The Week
Editorial Note: Healthcare Products LLC reviews the news wires looking for press releases and current articles relating to dementia. We write a brief description of each article and by clicking on its heading will bring you to the originally written story ...hope you enjoy The Alzheimer's News...
(Source: Mercola.com)-In the United States, Alzheimer's disease is currently at epidemic proportions, with 5.4 million Americans—including one in eight people aged 65 and over—living with the disease, according to the Alzheimer's Association's 2011 Alzheimer's Disease Facts and Figures.
By 2050, this is expected to jump to 16 million, and in the next 20 years, it is projected that Alzheimer's will affect one in four Americans, rivaling the current prevalence of obesity and diabetes.
There is still no known cure for this devastating disease, and very few treatments. Alzheimer's drugs are often of little to no benefit at all, which underscores the importance of prevention throughout your lifetime.
Research repeatedly suggests the best hope for patients lies in prevention through optimal diet, exercise and staying socially and mentally active. As recently reported by Forbes:
“[A] new study in Science suggested that last year’s ‘breakthrough’ pharmaceutical, bexarotene (Targretin) – a cancer drug that had initially received wide publicity for helping break up the plaques in Alzheimer’s – doesn’t seem to do this very well at all, and can have significant adverse side effects for the patient.
‘Something happened in that initial report – either something technically or otherwise, which we can’t put our hands on at this point in time,” study author Sangram Sisodia told US News & World Report. ‘Something is seriously wrong.’
(Source: CNN Health) - Wallack was 6, his great-grandmother Gertrude Finkelstein -- Great Grams as he called her -- was diagnosed with Alzheimer's disease. His parents were open with him, explaining in easy-to-understand terms that the illness would have a significant impact on her memory.
Wallack and his great-grandmother had always been close, but the debilitating disease took its toll on their relationship. She had cared for him when he was young; now he was one of her primary caregivers.
Wallack spent a large part his childhood watching over Great Grams, who was in and out of nursing homes until she moved in with his family. Despite his age, Wallack often held the responsibility of taking care of his great-grandmother alone when his parents could not.
He called it "bubby-sitting."
Alzheimer's affects 5.2 million Americans and millions more who are caregivers devoting their lives to helping affected loved ones. Caring for someone with Alzheimer's is no simple task. Symptoms include memory loss, difficulty in performing everyday tasks and not recognizing close family members.
"There were some hard times," says Wallack, now 17. He recalls a vacation to Hawaii when Great Grams grew fearful of the family, even though they were only looking out for her.
(Source: Science Daily) - Scientists at Washington University School of Medicine in St. Louis have found a way that corrupted, disease-causing proteins spread in the brain, potentially contributing to Alzheimer's disease, Parkinson's disease and other brain-damaging disorders.
The research identifies a specific type of receptor and suggests that blocking it may aid treatment of theses illnesses. The receptors are called heparan sulfate proteoglycans (HSPGs).
"Many of the enzymes that create HSPGs or otherwise help them function are good targets for drug treatments," said senior author Marc I. Diamond, MD, the David Clayson Professor of Neurology. "We ultimately should be able to hit these enzymes with drugs and potentially disrupt several neurodegenerative conditions."
The study is available online in the Proceedings of the National Academy of Sciences.
Over the last decade, Diamond has gathered evidence that Alzheimer's disease and other neurodegenerative diseases spread through the brain in a fashion similar to conditions such as mad cow disease, which are caused by misfolded proteins known as prions.
Proteins are long chains of amino acids that perform many basic biological functions. A protein's abilities are partially determined by the way it folds into a 3-D shape. Prions are proteins that have become folded in a fashion that makes them harmful.
Prions spread across the brain by causing other copies of the same protein to misfold.
Among the most infamous prion diseases are mad cow disease, which rapidly destroys the brain in cows, and a similar, inherited condition in humans called Creutzfeldt-Jakob disease.
(Source: Medical News Today) -The ability to draw spontaneously as well as from memory may be preserved in the brains of artists long after the deleterious effects of vascular dementia have diminished their capacity to complete simple, everyday tasks, according to a new study by physicians at St. Michael's Hospital.
The finding, in the Canadian Journal of Neurological Sciences, looked at the last few years of the late Mary Hecht, an internationally renowned sculptor, who was able to draw spur-of-the moment and detailed sketches of faces and figures, including from memory, despite an advanced case of vascular dementia.
"Art opens the mind," said Dr. Luis Fornazzari, neurological consultant at St. Michael's Hospital's Memory Clinic and lead author of the paper. "Mary Hecht was a remarkable example of how artistic abilities are preserved in spite of the degeneration of the brain and a loss in the more mundane, day-to-day memory functions."
Hecht, who died in April 2013 at 81, had been diagnosed with vascular dementia and was wheelchair-bound due to previous strokes. Despite her vast knowledge of art and personal talent, she was unable to draw the correct time on a clock, name certain animals or remember any of the words she was asked to recall.
But she quickly sketched an accurate portrait of a research student from the Memory Clinic. And she was able to draw a free-hand sketch of a lying Buddha figurine and reproduce it from memory a few minutes later. To the great delight of St. Michael's doctors, Hecht also drew an accurate sketch of famed cellist Mstislav Rostropovich after she learned of his death earlier that day on the radio.
The Alzheimer's Store Featured Product!
Item #0088 Handyman's Box
The Handyman's Box is a unique activity product - a source of pride, fascination and fun!
Recipe Of The Week
Warm wedges of this tasty quiche are packed with convenient frozen spinach, sweet red pepper and Swiss cheese. You can saute the bacon mixture the night before to reduce prep time on busy mornings.
Spinach Swiss Quiche
1 refrigerated pie pastry
4 turkey bacon strips, diced
1/4 cup chopped onion
1/4 cup chopped sweet red pepper
1 package (10 ounces) frozen chopped spinach, thawed and squeezed dry
2 cups egg substitute
1/2 cup fat-free cottage cheese
1/4 cup shredded reduced-fat Swiss cheese
1/2 teaspoon dried oregano
1/4 teaspoon dried parsley flakes
1/4 teaspoon each salt, pepper and paprika
6 tablespoons fat-free sour cream
On a lightly floured surface, unroll pastry. Transfer to a 9-in. pie plate. Trim pastry to 1/2 in. beyond edge of plate; flute edges. Line unpricked pastry with a double thickness of heavy-duty foil.
Bake at 450° for 8 minutes. Remove foil; bake 5 minutes longer. Cool on a wire rack. Reduce heat to 350°.
In a small skillet, cook the bacon, onion and red pepper until vegetables are tender; drain. Stir in spinach. Spoon spinach mixture into pastry.
In a small bowl, combine the egg substitute, cottage cheese, Swiss cheese and seasonings; pour over spinach mixture.
Bake for 35-40 minutes or until a knife inserted near the center comes out clean. Let stand for 10 minutes before cutting.
Serve with sour cream. Yield: 6 servings.
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